CELL CALCIUM
ISSN:0143-4160

CELL CALCIUM

CELL CALCIUM
学科领域:生物学
是否预警:不在预警名单内
是否OA:
录用周期:平均3月
新锐分区:生物学2区
年发文量:58
影响因子:4
JCR分区:Q1

基本信息

细胞钙涵盖了生命系统中的钙代谢和信号转导领域,从无机化学、生理学、分子生物学和病理学等方面进行研究。钙在细胞凋亡、坏死和细胞器重构等细胞事件中的调节作用钙调节对健康和疾病结局的影响
0143-4160SCIE/Scopus收录
4
4.6
2026年3月发布
点击查看历史分区趋势    >
大类学科小类学科Top期刊综述期刊
生物学2区
CELL BIOLOGY 细胞生物学
3区
PHYSIOLOGY 生理学
2区
N/A
WOS期刊SCI分区  2024-2025最新升级版
按JIF指标学科分区收集子录JIF分区JIF排名百分位
学科:CELL BIOLOGY
SCIE
Q2
90/204
学科:PHYSIOLOGY
SCIE
Q1
14/87
按JCR指标学科分区收集子录JCR分区JCR排名百分位
学科:CELL BIOLOGY
SCIE
Q1
46/204
学科:PHYSIOLOGY
SCIE
Q1
15/87
107
58
6%约80%平均3月-生物-细胞生物学
5%
时间预警情况
2026年03月发布的新锐学术版不在预警名单中
2025年03月发布的2025版不在预警名单中
2024年02月发布的2024版不在预警名单中
2023年01月发布的2023版不在预警名单中
2021年12月发布的2021版不在预警名单中
2020年12月发布的2020版不在预警名单中
93.10%37.16%1.05%
CiteScore:8.30
SJR:1.531
SNIP:0.888
学科类别分区排名百分位
大类:Biochemistry, Genetics and Molecular Biology
小类:Physiology
Q1
30 / 190
大类:Biochemistry, Genetics and Molecular Biology
小类:Molecular Biology
Q2
108 / 410
大类:Biochemistry, Genetics and Molecular Biology
小类:Cell Biology
Q2
86 / 285

期刊高被引文献

Spatiotemporal pattern of calcium activity in astrocytic network.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2018.12.007
Mechanosensitive ion channels push cancer progression.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.03.007
Numbers count: How STIM and Orai stoichiometry affect store-operated calcium entry.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.02.002
CRAC channel regulation of innate immune cells in health and disease.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.01.003
A novel STIM1-Orai1 gating interface essential for CRAC channel activation.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.02.009
Store-operated calcium entry in thrombosis and thrombo-inflammation.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2018.11.005
On the special role of NCX in astrocytes: Translating Na+-transients into intracellular Ca2+ signals.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.102154
ORAI channels in cellular remodeling of cardiorespiratory disease.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.01.005
SOCE and STIM1 signaling in the heart: Timing and location matter.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2018.11.008
Tau inhibits mitochondrial calcium efflux and makes neurons vulnerable to calcium-induced cell death.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.102150
Pseudomonas aeruginosa β-carbonic anhydrase, psCA1, is required for calcium deposition and contributes to virulence.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.102080
ER Ca2+ release and store-operated Ca2+ entry - partners in crime or independent actors in oncogenic transformation?
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.102061
The structure of TRPC ion channels.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.03.005
Structural biology of thermoTRPV channels.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.102106
Extracellular calcium stimulates osteogenic differentiation of human adipose-derived stem cells by enhancing bone morphogenetic protein-2 expression.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.102058
Does stromal interaction molecule-1 have five senses?
来源期刊:Cell calciumDOI:10.1016/j.ceca.2018.12.001
Dysregulation of Mg2+ homeostasis contributes to acquisition of cancer hallmarks.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.102078
Methylglyoxal evokes acute Ca2+ transients in distinct cell types and increases agonist-evoked Ca2+ entry in endothelial cells via CRAC channels.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.01.002
L-type calcium channel modulates mechanosensitivity of the cardiomyocyte cell line H9c2.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.02.008
Na+-dependent transporters: The backbone of astroglial homeostatic function.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.102136
Getting close. Lysosome-ER contact sites tailor Ca2+ signals.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.02.003
Role of Cl- channels in primary brain tumour.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.05.004
Discovery and characterization of a positive allosteric modulator of transient receptor potential canonical 6 (TRPC6) channels.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2018.12.009
A single point mutation in the TRPC3 lipid-recognition window generates supersensitivity to benzimidazole channel activators.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.02.007
Basic and editing mechanisms underlying ion transport and regulation in NCX variants.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.102131
Physiological functions of mitochondrial Na+-Ca2+ exchanger, NCLX, in lymphocytes.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.102114
Potassium-dependent sodium-calcium exchanger (NCKX) isoforms and neuronal function.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.102135
Focus on TRP channels in cystic fibrosis.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.05.007
CRAC channels in secretory epithelial cell function and disease.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2018.12.010
Ion occupancy of the channel pore is critical for triggering excitation-transcription (ET) coupling.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.102102
Control of cardiac contraction by sodium: Promises, reckonings, and new beginnings.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.102129
Microcalcifications, calcium-sensing receptor, and cancer.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.06.005
Involvement of N-type Ca2+ channel in microglial activation and its implications to aging-induced exaggerated cytokine response.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.102059
Oxytocin induces intracellular Ca2+ release in cardiac fibroblasts from neonatal rats.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.102099
Sodium pumps, ouabain and aldosterone in the brain: A neuromodulatory pathway underlying salt-sensitive hypertension and heart failure.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.102151
Extracellular mild acidosis decreases the Ca2+ permeability of the human NMDA receptors.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.04.001
Avoiding raising the ire of IRE1α.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.102056
Splice-variant-specific effects of primary aldosteronism point mutations on human CaV3.2 calcium channels.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.102104
NCX activity generates spontaneous Ca2+ oscillations in the astrocytic leaflet microdomain.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.102137
Unexpected Ca2+-mobilization of oxaliplatin via H1 histamine receptors.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.102128
Structural insights into group II TRP channels.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.102107
Noise analysis of cytosolic calcium image data.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.102152
Effects of his-tags on physical properties of parvalbumins.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2018.11.006
L-asparaginase-induced apoptosis in ALL cells involves IP3 receptor signaling.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.102076
Structure-function relationships of K+-dependent Na+/Ca2+ exchangers (NCKX).
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.102153
Nuclear localization of NCX: Role in Ca2+ handling and pathophysiological implications.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.102143
Cytosolic and mitochondrial Ca2+ concentrations in primary hepatocytes change with ageing and in consequence of an mtDNA mutation.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.102055
The emerging link between IP3 receptor turnover and Hereditary Spastic Paraplegia.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.102142
Mitochondrial Ca2+ laMety directs fibrosis.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.102155
Recent progress in structural studies on canonical TRP ion channels.
来源期刊:Cell calciumDOI:10.1016/j.ceca.2019.102075

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