The interplay of aging, genetics and environmental factors in the pathogenesis of Parkinson’s disease
来源期刊:Translational NeurodegenerationDOI:10.1186/s40035-019-0165-9
Blocking meningeal lymphatic drainage aggravates Parkinson’s disease-like pathology in mice overexpressing mutated α-synuclein
来源期刊:Translational NeurodegenerationDOI:10.1186/s40035-019-0147-y
Exploratory study on microRNA profiles from plasma-derived extracellular vesicles in Alzheimer’s disease and dementia with Lewy bodies
来源期刊:Translational NeurodegenerationDOI:10.1186/s40035-019-0169-5
Beyond the synucleinopathies: alpha synuclein as a driving force in neurodegenerative comorbidities
来源期刊:Translational NeurodegenerationDOI:10.1186/s40035-019-0172-x
Efficient RT-QuIC seeding activity for α-synuclein in olfactory mucosa samples of patients with Parkinson’s disease and multiple system atrophy
来源期刊:Translational NeurodegenerationDOI:10.1186/s40035-019-0164-x
Current progress of mitochondrial transplantation that promotes neuronal regeneration
来源期刊:Translational NeurodegenerationDOI:10.1186/s40035-019-0158-8
Constipation, deficit in colon contractions and alpha-synuclein inclusions within the colon precede motor abnormalities and neurodegeneration in the central nervous system in a mouse model of alpha-synucleinopathy
来源期刊:Translational NeurodegenerationDOI:10.1186/s40035-019-0146-z
Parkinson’s disease in China: a forty-year growing track of bedside work
来源期刊:Translational NeurodegenerationDOI:10.1186/s40035-019-0162-z
Erythrocytic α-Synuclein as a potential biomarker for Parkinson’s disease
来源期刊:Translational NeurodegenerationDOI:10.1186/s40035-019-0155-y
Microbiome changes: an indicator of Parkinson’s disease?
来源期刊:Translational NeurodegenerationDOI:10.1186/s40035-019-0175-7
Targeting γ-secretase triggers the selective enrichment of oligomeric APP-CTFs in brain extracellular vesicles from Alzheimer cell and mouse models
来源期刊:Translational NeurodegenerationDOI:10.1186/s40035-019-0176-6
Detecting neurodegenerative pathology in multiple sclerosis before irreversible brain tissue loss sets in
来源期刊:Translational NeurodegenerationDOI:10.1186/s40035-019-0178-4
Longitudinal diffusion tensor magnetic resonance imaging analysis at the cohort level reveals disturbed cortical and callosal microstructure with spared corticospinal tract in the TDP-43G298S ALS mouse model
来源期刊:Translational NeurodegenerationDOI:10.1186/s40035-019-0163-y
A longitudinal assessment of retinal function and structure in the APP/PS1 transgenic mouse model of Alzheimer’s disease
来源期刊:Translational NeurodegenerationDOI:10.1186/s40035-019-0170-z
Dihydromyricetin and Salvianolic acid B inhibit alpha-synuclein aggregation and enhance chaperone-mediated autophagy
来源期刊:Translational NeurodegenerationDOI:10.1186/s40035-019-0159-7
Cognitive decline is related to high blood glucose levels in older Chinese adults with the ApoE ε3/ε3 genotype
来源期刊:Translational NeurodegenerationDOI:10.1186/s40035-019-0151-2
Molecular targets for modulating the protein translation vital to proteostasis and neuron degeneration in Parkinson’s disease
来源期刊:Translational NeurodegenerationDOI:10.1186/s40035-019-0145-0
DNA repair deficiency in neuropathogenesis: when all roads lead to mitochondria
来源期刊:Translational NeurodegenerationDOI:10.1186/s40035-019-0156-x
Alzheimer’s in a dish – induced pluripotent stem cell-based disease modeling
来源期刊:Translational NeurodegenerationDOI:10.1186/s40035-019-0161-0
No association between Parkinson disease and autoantibodies against NMDA-type glutamate receptors
来源期刊:Translational NeurodegenerationDOI:10.1186/s40035-019-0153-0
Lower serum expression of miR-181c-5p is associated with increased plasma levels of amyloid-beta 1–40 and cerebral vulnerability in normal aging
来源期刊:Translational NeurodegenerationDOI:10.1186/s40035-019-0174-8
Xenografting of human umbilical mesenchymal stem cells from Wharton’s jelly ameliorates mouse spinocerebellar ataxia type 1
来源期刊:Translational NeurodegenerationDOI:10.1186/s40035-019-0166-8
NF-κB/c-Rel deficiency causes Parkinson’s disease-like prodromal symptoms and progressive pathology in mice
来源期刊:Translational NeurodegenerationDOI:10.1186/s40035-019-0154-z
Clinical features and genetic spectrum in Chinese patients with recessive hereditary spastic paraplegia
来源期刊:Translational NeurodegenerationDOI:10.1186/s40035-019-0157-9
Enriched gestation activates the IGF pathway to evoke embryo-adult benefits to prevent Alzheimer’s disease
来源期刊:Translational NeurodegenerationDOI:10.1186/s40035-019-0149-9
Oral administration of the cannabigerol derivative VCE-003.2 promotes subventricular zone neurogenesis and protects against mutant huntingtin-induced neurodegeneration
来源期刊:Translational NeurodegenerationDOI:10.1186/s40035-019-0148-x
Bee venom phospholipase A2 ameliorates amyloidogenesis and neuroinflammation through inhibition of signal transducer and activator of transcription-3 pathway in Tg2576 mice
来源期刊:Translational NeurodegenerationDOI:10.1186/s40035-019-0167-7
Clinicopathologic characterization and abnormal autophagy of CSF1R-related leukoencephalopathy
来源期刊:Translational NeurodegenerationDOI:10.1186/s40035-019-0171-y
Oscillation-specific nodal alterations in early to middle stages Parkinson’s disease
来源期刊:Translational NeurodegenerationDOI:10.1186/s40035-019-0177-5
Transplantation of bone marrow derived macrophages reduces markers of neuropathology in an APP/PS1 mouse model
来源期刊:Translational NeurodegenerationDOI:10.1186/s40035-019-0173-9
How to spot ocular abnormalities in progressive supranuclear palsy? A practical review
来源期刊:Translational NeurodegenerationDOI:10.1186/s40035-019-0160-1
Kallikrein-related peptidases 6 and 10 are elevated in cerebrospinal fluid of patients with Alzheimer’s disease and associated with CSF-TAU and FDG-PET
来源期刊:Translational NeurodegenerationDOI:10.1186/s40035-019-0168-6
Correction to: Better survival in female SOD1-mutant patients with ALS: a study of SOD1-related natural history
来源期刊:Translational NeurodegenerationDOI:10.1186/s40035-019-0150-3