LncRNAs in vascular biology and disease.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2018.01.003
Foam cell formation: A new target for fighting atherosclerosis and cardiovascular disease.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2018.08.002
Randomized controlled trial of rivaroxaban versus warfarin in the management of acute non-neoplastic portal vein thrombosis.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2018.05.002
Non-coding RNA regulation of endothelial and macrophage functions during atherosclerosis.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2018.03.001
MicroRNA-221-3p promotes pulmonary artery smooth muscle cells proliferation by targeting AXIN2 during pulmonary arterial hypertension.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2017.07.002
The function of miR-143, miR-145 and the MiR-143 host gene in cardiovascular development and disease
来源期刊:Vascular PharmacologyDOI:10.1016/j.vph.2018.11.006
Uric acid: from a biological advantage to a potential danger. A focus on cardiovascular effects.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2019.106565
Long non-coding RNAs in vascular biology and disease.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2018.03.003
Non-coding RNAs in vascular remodeling and restenosis.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2018.10.006
The function of long non-coding RNAs in vascular biology and disease.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2018.06.004
Linking diabetic vascular complications with LncRNAs.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2018.01.007
Non-coding RNAs in lipid metabolism.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2018.06.011
Pharmacological strategies to inhibit intra-plaque angiogenesis in atherosclerosis.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2018.06.014
Adenosine receptor agonists deepen the inhibition of platelet aggregation by P2Y12 antagonists.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2018.11.005
Beyond endothelial cells: Vascular endothelial growth factors in heart, vascular anomalies and placenta.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2018.10.005
Amorphous nano-selenium quantum dots improve endothelial dysfunction in rats and prevent atherosclerosis in mice through Na+/H+ exchanger 1 inhibition.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2019.01.005
Oxidative stress and vascular stiffness in hypertension: A renewed interest for antioxidant therapies?
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2019.03.004
Asiatic acid stabilizes cytoskeletal proteins and prevents TNF-α-induced disorganization of cell-cell junctions in human aortic endothelial cells.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2018.08.005
Vascular angiotensin AT1 receptor neuromodulation in fetal programming of hypertension.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2018.10.003
Matrix metalloproteinase (MMP)-2 activation by oxidative stress decreases aortic calponin-1 levels during hypertrophic remodeling in early hypertension.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2018.10.002
Cost effectiveness of lifelong therapy with PCSK9 inhibitors for lowering cardiovascular events in patients with stable coronary artery disease: Insights from the Ludwigshafen Risk and Cardiovascular Health cohort.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2019.106566
Alogliptin improves endothelial function by promoting autophagy in perivascular adipose tissue of obese mice through a GLP-1-dependent mechanism.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2018.11.003
Non-coding RNAs in aneurysmal aortopathy.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2018.06.008
Switching between P2Y12 antagonists - From bench to bedside.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2019.01.003
Modulation of the monocyte/macrophage system in heart failure by targeting heme oxygenase-1.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2018.08.011
Inflammation and myointimal hyperplasia. Correlation with hemodynamic forces.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2017.06.001
CRISPR links to long noncoding RNA function in mice: A practical approach.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2019.02.004
Treatment of cyclosporine induced hypertension: Results from a long-term observational study using different antihypertensive medications.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2018.06.012
Long non-coding RNA-SRA promotes neointimal hyperplasia and vascular smooth muscle cells proliferation via MEK-ERK-CREB pathway.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2019.02.005
Vitamin D levels and platelet reactivity in diabetic patients receiving dual antiplatelet therapy.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2019.106564
The endothelial status reflected by circulating endothelial cells, circulating endothelial progenitor cells and soluble thrombomodulin in patients with mild and resistant hypertension.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2018.12.005
Ex vivo microangioCT: Advances in microvascular imaging.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2018.09.003
HMGB1 enhances AGE-mediated VSMC proliferation via an increase in 5-LO-linked RAGE expression.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2019.04.001
Drug-induced hypertension: Know the problem to know how to deal with it.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2019.02.002
Role of amino acid metabolism in angiogenesis.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2018.11.001
Variety matters: Diverse functions of monocyte subtypes in vascular inflammation and atherogenesis.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2018.12.002
Microbiota are critical for vascular physiology: Germ-free status weakens contractility and induces sex-specific vascular remodeling in mice.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2019.106633
SNF472, a novel anti-crystallization agent, inhibits induced calcification in an in vitro model of human aortic valve calcification.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2019.106583
Stox1 induced the proliferation and cell cycle arrest in pulmonary artery smooth muscle cells via AKT signaling pathway.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2019.106568
Effects of sildenafil on maximum walking time in patients with arterial claudication: The ARTERIOFIL study.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2019.05.003
Long non-coding RNAs: A crucial part of the vasculature puzzle.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2018.02.008
Everolimus depletes plaque macrophages, abolishes intraplaque neovascularization and improves survival in mice with advanced atherosclerosis.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2018.12.004
Rapid effects of aldosterone on platelets, coagulation, and fibrinolysis lead to experimental thrombosis augmentation.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2019.106598
Elevated luteinizing hormone contributes to atherosclerosis formation by inhibiting nitric oxide synthesis via PI3K/Akt pathway.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2019.106582
Lipid core nanoparticles as vehicle for docetaxel reduces atherosclerotic lesion, inflammation, cell death and proliferation in an atherosclerosis rabbit model.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2019.02.003
LIM-only protein FHL2 attenuates inflammation in vascular smooth muscle cells through inhibition of the NFκB pathway.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2019.106634
GLP-1-based therapies to boost autophagy in cardiometabolic patients: From experimental evidence to clinical trials.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2019.03.003
Atorvastatin enhances endothelial adherens junctions through promoting VE-PTP gene transcription and reducing VE-cadherin-Y731 phosphorylation.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2018.06.003
Antiretroviral drug-induced endothelial dysfunction is improved by dual PPARα/γ stimulation in obesity.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2019.106577
Lessons learned from a lncRNA odyssey for two genes with vascular functions, DLL4 and TIE1.
来源期刊:Vascular pharmacologyDOI:10.1016/j.vph.2018.06.010