Defining inflammatory cell states in rheumatoid arthritis joint synovial tissues by integrating single-cell transcriptomics and mass cytometry
来源期刊:Nature immunologyDOI:10.1038/s41590-019-0378-1
Interferon target-gene expression and epigenomic signatures in health and disease
来源期刊:Nature ImmunologyDOI:10.1038/s41590-019-0466-2
The lung environment controls alveolar macrophage metabolism and responsiveness in type 2 inflammation
来源期刊:Nature ImmunologyDOI:10.1038/s41590-019-0352-y
c-Maf-dependent Treg cell control of intestinal TH17 cells and IgA establishes host–microbiota homeostasis
来源期刊:Nature ImmunologyDOI:10.1038/s41590-019-0316-2
Inflammatory macrophage dependence on NAD+ salvage is a consequence of reactive oxygen species–mediated DNA damage
来源期刊:Nature ImmunologyDOI:10.1038/s41590-019-0336-y
Mitochondrial fragmentation limits NK cell-based tumor immunosurveillance
来源期刊:Nature ImmunologyDOI:10.1038/s41590-019-0511-1
The transcription factor c-Myb regulates CD8+ T cell stemness and antitumor immunity
来源期刊:Nature immunologyDOI:10.1038/s41590-018-0311-z
Cryptic activation of an Irf8 enhancer governs cDC1 fate specification
来源期刊:Nature immunologyDOI:10.1038/s41590-019-0450-x
Flip the coin: IL-7 and IL-7R in health and disease
来源期刊:Nature ImmunologyDOI:10.1038/s41590-019-0479-x
IL-17 receptor–based signaling and implications for disease
来源期刊:Nature ImmunologyDOI:10.1038/s41590-019-0514-y
Endogenous oxidized phospholipids reprogram cellular metabolism and boost hyperinflammation
来源期刊:Nature immunologyDOI:10.1038/s41590-019-0539-2
The transcription factor TCF-1 enforces commitment to the innate lymphoid cell lineage
来源期刊:Nature immunologyDOI:10.1038/s41590-019-0445-7
Uncoupling protein 2 reprograms the tumor microenvironment to support the anti-tumor immune cycle
来源期刊:Nature ImmunologyDOI:10.1038/s41590-018-0290-0
TSLP: from allergy to cancer
来源期刊:Nature ImmunologyDOI:10.1038/s41590-019-0524-9
TCR sequencing paired with massively parallel 3′ RNA-seq reveals clonotypic T cell signatures
来源期刊:Nature ImmunologyDOI:10.1038/s41590-019-0544-5
Basophil-derived tumor necrosis factor can enhance survival in a sepsis model in mice
来源期刊:Nature immunologyDOI:10.1038/s41590-018-0288-7
RBPJ-dependent Notch signaling initiates the T cell program in a subset of thymus-seeding progenitors
来源期刊:Nature immunologyDOI:10.1038/s41590-019-0518-7
Quantifying in situ adaptive immune cell cognate interactions in humans
来源期刊:Nature immunologyDOI:10.1038/s41590-019-0315-3
Safeguard function of PU.1 shapes the inflammatory epigenome of neutrophils
来源期刊:Nature ImmunologyDOI:10.1038/s41590-019-0343-z
SDHA gain-of-function engages inflammatory mitochondrial retrograde signaling via KEAP1–Nrf2
来源期刊:Nature ImmunologyDOI:10.1038/s41590-019-0482-2
A temporal thymic selection switch and ligand binding kinetics constrain neonatal Foxp3+Tregcell development.
来源期刊:Nature ImmunologyDOI:10.2210/PDB6MNO/PDB
Cytokines in the balance
来源期刊:Nature ImmunologyDOI:10.1038/s41590-019-0557-0
Author Correction: Mosaic nanoparticle display of diverse influenza virus hemagglutinins elicits broad B cell responses
来源期刊:Nature ImmunologyDOI:10.1038/s41590-019-0395-0
Tissue adaptation
来源期刊:Nature ImmunologyDOI:10.1038/s41590-018-0306-9
At the end of the beginning: immunotherapies as living drugs
来源期刊:Nature ImmunologyDOI:10.1038/s41590-019-0452-8
CD4+ T cell signatures in HIV infection
来源期刊:Nature ImmunologyDOI:10.1038/s41590-019-0447-5
The IFN-λ Pony Express
来源期刊:Nature ImmunologyDOI:10.1038/s41590-019-0362-9
NAD-biosynthetic pathways regulate innate immunity
来源期刊:Nature ImmunologyDOI:10.1038/s41590-019-0353-x
Metabolic requirements for expanding and arming a clone army
来源期刊:Nature ImmunologyDOI:10.1038/s41590-018-0299-4
IL-15 in autoimmunity and cancer: O-tu-b or not O-tu-b?
来源期刊:Nature ImmunologyDOI:10.1038/s41590-019-0424-z
Author Correction: Subsets of exhausted CD8+ T cells differentially mediate tumor control and respond to checkpoint blockade
来源期刊:Nature ImmunologyDOI:10.1038/s41590-019-0528-5
Humidity and immunity
来源期刊:Nature ImmunologyDOI:10.1038/s41590-019-0434-x
Too much can be as bad as too little
来源期刊:Nature ImmunologyDOI:10.1038/s41590-019-0498-7
γδ T cells support learning
来源期刊:Nature ImmunologyDOI:10.1038/s41590-019-0546-3
Human and translational immunology in the third millennium: progress, challenges and opportunities
来源期刊:Nature ImmunologyDOI:10.1038/s41590-019-0543-6
Neutralizing HSV
来源期刊:Nature ImmunologyDOI:10.1038/s41590-019-0534-7
Histone lactylation
来源期刊:Nature ImmunologyDOI:10.1038/s41590-019-0551-6
Protecting baby
来源期刊:Nature ImmunologyDOI:10.1038/s41590-019-0460-8
Author Correction: c-Kit-positive ILC2s exhibit an ILC3-like signature that may contribute to IL-17-mediated pathologies
来源期刊:Nature ImmunologyDOI:10.1038/s41590-019-0554-3
Protective Ig responses
来源期刊:Nature ImmunologyDOI:10.1038/s41590-019-0437-7
cDC2 subsets
来源期刊:Nature ImmunologyDOI:10.1038/s41590-019-0552-5
Treg cells mend a broken heart
来源期刊:Nature ImmunologyDOI:10.1038/s41590-019-0547-2
DCs remember
来源期刊:Nature ImmunologyDOI:10.1038/s41590-019-0486-y
Early life encounters
来源期刊:Nature ImmunologyDOI:10.1038/s41590-019-0553-4
Author Correction: ‘Stealth’ corporate innovation: an emerging threat for therapeutic drug development
来源期刊:Nature ImmunologyDOI:10.1038/s41590-019-0531-x
Food tolerance
来源期刊:Nature ImmunologyDOI:10.1038/s41590-019-0431-0
How to wake a killer
来源期刊:Nature ImmunologyDOI:10.1038/s41590-019-0541-8
Suppressing alarmins
来源期刊:Nature ImmunologyDOI:10.1038/s41590-018-0308-7
Vaccine sex differences
来源期刊:Nature ImmunologyDOI:10.1038/s41590-018-0310-0
GC B cells ‘AKT’ to blunt BCR signaling
来源期刊:Nature ImmunologyDOI:10.1038/s41590-019-0384-3