JOURNAL OF ENDOCRINOLOGY
ISSN:0022-0795

JOURNAL OF ENDOCRINOLOGY

J ENDOCRINOL
学科领域:医学
是否预警:不在预警名单内
是否OA:
录用周期:偏慢,4-8周
新锐分区:医学3区
年发文量:76
影响因子:3.9
JCR分区:Q2

基本信息

《内分泌学杂志》是一本全球领先的杂志,发表原创研究文章、评论和科学指南。它的重点是内分泌生理学和代谢,包括激素分泌;激素作用;生物效应。该杂志发表器官、组织和整个生物体水平的基础和转化研究。
0022-0795SCIE/Scopus收录
3.9
5.4
2026年3月发布
点击查看历史分区趋势    >
大类学科小类学科Top期刊综述期刊
医学3区
ENDOCRINOLOGY & METABOLISM 内分泌学与代谢
3区
N/A
WOS期刊SCI分区  2024-2025最新升级版
按JIF指标学科分区收集子录JIF分区JIF排名百分位
学科:ENDOCRINOLOGY & METABOLISM
SCIE
Q2
56/193
按JCR指标学科分区收集子录JCR分区JCR排名百分位
学科:ENDOCRINOLOGY & METABOLISM
SCIE
Q2
69/193
132
76
13%0偏慢,4-8周-医学-内分泌学与代谢
0%
时间预警情况
2026年03月发布的新锐学术版不在预警名单中
2025年03月发布的2025版不在预警名单中
2024年02月发布的2024版不在预警名单中
2023年01月发布的2023版不在预警名单中
2021年12月发布的2021版不在预警名单中
2020年12月发布的2020版不在预警名单中
51.32%27.52%-
CiteScore:6.90
SJR:1.194
SNIP:1.029
学科类别分区排名百分位
大类:Medicine
小类:Endocrinology, Diabetes and Metabolism
Q2
64 / 247
大类:Medicine
小类:Endocrinology
Q2
43 / 130

期刊高被引文献

Epigenetic responses and the developmental origins of health and disease.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0009
Inhibitors of apoptosis protect the ovarian reserve from cyclophosphamide.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0370
Nutritional adversity, sex and reproduction: 30 years of DOHaD and what have we learned?
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0048
Epidemiological evidence for the developmental origins of health and disease: effects of prenatal undernutrition in humans.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0683
Leptin resensitisation: a reversion of leptin-resistant states.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0606
Developmental programming of the HPA axis and related behaviours: epigenetic mechanisms.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0057
Central growth hormone signaling is not required for the timing of puberty.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0242
Nrf2 represses the onset of type 1 diabetes in non-obese diabetic mice.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0355
Impact of exenatide on mitochondrial lipid metabolism in mice with nonalcoholic steatohepatitis.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0007
Maternal vitamin D deficiency and developmental origins of health and disease (DOHaD).
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0541
Terpenes, hormones, and life: isoprene rule revisited.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0084
Effect of exercise and butyrate supplementation on microbiota composition and lipid metabolism.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0122
Developmental pathways and programming of diabetes: epidemiological aspects.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0680
Mice lacking adenosine 2A receptor reveal increased severity of MCD-induced NASH.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0198
Metformin improves ovarian insulin signaling alterations caused by fetal programming.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0520
High glucose up-regulates osteopontin expression by FoxO1 activation in macrophage.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0594
Thyroid hormone signalling and consequences for cardiac development.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0704
The glucocorticoid receptor in osteoprogenitors regulates bone mass and marrow fat.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0230
The depot-specific and essential roles of CBP/p300 in regulating adipose plasticity.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0361
Telmisartan prevents development of obesity and normalizes hypothalamic lipid droplets.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0319
Voluntary exercise is motivated by ghrelin, possibly related to the central reward circuit
来源期刊:The Journal of EndocrinologyDOI:10.1530/JOE-19-0213
Chronic predator stress in female mice reduces primordial follicle numbers: implications for the role of ghrelin.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0109
Identification of circular RNAs in the immature and mature rat anterior pituitary.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0540
Antenatal dexamethasone treatment transiently alters diastolic function in the mouse fetal heart
来源期刊:The Journal of EndocrinologyDOI:10.1530/JOE-18-0666
Mechanism of SEMA3G knockdown-mediated attenuation of high-fat diet-induced obesity.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0029
High glucose alters fetal rat islet transcriptome and induces progeny islet dysfunction.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0493
Vagal afferents contribute to sympathoexcitation-driven metabolic dysfunctions.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0623
Comparing methods to normalize insulin secretion shows the process may not be needed.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0542
L- and T-type calcium channels control aldosterone production from human adrenals.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0259
SRT1720 retards renal fibrosis via inhibition of HIF1α /GLUT1 in diabetic nephropathy.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0536
Testosterone inhibits aneurysm formation and vascular inflammation in male mice.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0646
Anti-insulin resistance effects of salidroside through mitochondrial quality control.
来源期刊:The Journal of endocrinologyDOI:10.1530/joe-19-0393
Prenatal ethanol exposure induces susceptibility to premature ovarian insufficiency.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0063
BMP7 improves insulin signal transduction in the liver.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0693
Role of ovarian sympathetic nerves and cholinergic local system during cold stress.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0125
CaV1.2 and CaV1.3 voltage-gated L-type Ca2+ channels in rat white fat adipocytes.
来源期刊:The Journal of endocrinologyDOI:10.1530/joe-19-0493
Role of neutrophil extracellular traps in chronic kidney injury induced by bisphenol-A.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0608
Impaired LH surge amplitude in gonadotrope-specific progesterone receptor knockout mice.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0013
Cholestasis-associated glucocorticoid overexposure does not increase atherogenesis.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0079
Adhesion G-protein coupled receptor, GPR56, is required for Müllerian duct development in the chick.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0419
Monomethylfumarate protects against ovariectomy-related changes in body composition.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0691
Vincamine as a GPR40 agonist improves glucose homeostasis in type 2 diabetic mice.
来源期刊:The Journal of endocrinologyDOI:10.1530/joe-18-0432
Hyperinsulinemia restrains endometrial angiogenesis during decidualization in mice and human models.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0127
Hematopoiesis in aged female mice devoid of thyroid hormone receptors.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0339
Thermoneutrality improves skeletal impairment in adult Prader-Willi syndrome mice.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0279
Baicalin inhibits recruitment of GATA1 to the HSD3B2 promoter and reverses hyperandrogenism of PCOS.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0678
Selenoprotein S regulates adipogenesis through IRE1α-XBP1 pathway.
来源期刊:The Journal of endocrinologyDOI:10.1530/joe-19-0292
Glucagon regulates hepatic mitochondrial function and biogenesis through FOXO1.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0081
Forced internal desynchrony induces cardiometabolic alterations in adult rats.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0026
Inhibition of vascular adventitial remodeling by netrin-1 in diabetic rats.
来源期刊:The Journal of endocrinologyDOI:10.1530/joe-19-0258

相关文章

2026年3月发布
大类学科小类学科Top期刊综述期刊
医学3区
ENDOCRINOLOGY & METABOLISM 内分泌学与代谢
3区
N/A
2025年3月升级版
大类学科小类学科Top期刊综述期刊
医学3区
ENDOCRINOLOGY & METABOLISM 内分泌学与代谢
4区
2023年12月旧的升级版
大类学科小类学科Top期刊综述期刊
医学3区
ENDOCRINOLOGY & METABOLISM 内分泌学与代谢
3区