Circular RNA Ttc3 regulates cardiac function after myocardial infarction by sponging miR-15b.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.03.007
Mitochondrial LonP1 protects cardiomyocytes from ischemia/reperfusion injury in vivo.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2018.12.017
Gut microbe-derived metabolite trimethylamine N-oxide accelerates fibroblast-myofibroblast differentiation and induces cardiac fibrosis.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.07.004
p53 plays a crucial role in endothelial dysfunction associated with hyperglycemia and ischemia.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.02.010
DNA methyltransferase 1 and Krüppel-like factor 4 axis regulates macrophage inflammation and atherosclerosis.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.01.009
Cardiac repair and the putative role of stem cells.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.01.022
Cardiac-specific deficiency of the mitochondrial calcium uniporter augments fatty acid oxidation and functional reserve.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2018.12.019
Enhancement of human iPSC-derived cardiomyocyte maturation by chemical conditioning in a 3D environment.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.10.001
A short history of the development of mathematical models of cardiac mechanics
来源期刊:Journal of Molecular and Cellular CardiologyDOI:10.1016/j.yjmcc.2018.11.015
Oxidative stress contributes to vascular calcification in patients with chronic kidney disease.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.12.006
Ablation of the calpain-targeted site in cardiac myosin binding protein-C is cardioprotective during ischemia-reperfusion injury.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.03.006
Heparanase protects the heart against chemical or ischemia/reperfusion injury.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.04.008
Programmed death-ligand 1 triggers PASMCs pyroptosis and pulmonary vascular fibrosis in pulmonary hypertension.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.10.008
Early inhibition of endothelial retinoid uptake upon myocardial infarction restores cardiac function and prevents cell, tissue, and animal death.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2018.11.012
Crucial role of NLRP3 inflammasome in a murine model of Kawasaki disease.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.11.158
Pseudo-bipolar spindle formation and cell division in postnatal binucleated cardiomyocytes.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.07.005
Cardiac arrhythmias after renal I/R depend on IL-1β.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.04.025
HDAC inhibition as a therapeutic strategy in myocardial ischemia/reperfusion injury.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.02.013
Myocardial maladaptation to pressure overload in CB2 receptor-deficient mice.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.06.003
Exosomal non-coding RNAs (Exo-ncRNAs) in cardiovascular health.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.09.016
Phosphorylation of Cx43 residue Y313 by Src contributes to blocking the interaction with Drebrin and disassembling gap junctions.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2018.11.008
Dysregulation of IL-33/ST2 signaling and myocardial periarteriolar fibrosis.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.01.018
Requisite roles of LOX-1, JNK, and arginase in diabetes-induced endothelial vasodilator dysfunction of porcine coronary arterioles.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.04.015
Multiple roles of KLF15 in the heart: Underlying mechanisms and therapeutic implications.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.01.024
Activation of the calcium-sensing receptor in human valvular interstitial cells promotes calcification.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.01.021
Heterotypic docking compatibility of human connexin37 with other vascular connexins.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2018.12.013
Deficiency of NONO is associated with impaired cardiac function and fibrosis in mice.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.10.004
Smooth muscle-specific LKB1 deletion exaggerates angiotensin II-induced abdominal aortic aneurysm in mice.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.03.021
Sam68 impedes the recovery of arterial injury by augmenting inflammatory response.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.10.003
Hypoplastic left heart syndrome: From bedside to bench and back.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.08.005
Inhibition of DRG-TRPV1 upregulation in myocardial ischemia contributes to exogenous cardioprotection.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.12.003
Protective transcriptional mechanisms in cardiomyocytes and cardiac fibroblasts.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.04.023
In vitro and in vivo cardioprotective and metabolic efficacy of vitamin E TPGS/Apelin.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.12.001
MIR148A family regulates cardiomyocyte differentiation of human embryonic stem cells by inhibiting the DLL1-mediated NOTCH signaling pathway.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.06.014
Genetic and functional implications of an exonic TRIM55 variant in heart failure.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.12.008
Inhibition of Kir2.1 channel-induced depolarization promotes cell biological activity and differentiation by modulating autophagy in late endothelial progenitor cells.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2018.11.005
Mitochondrial thioredoxin-2 maintains HCN4 expression and prevents oxidative stress-mediated sick sinus syndrome.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.10.009
Large scale, unbiased analysis of elementary calcium signaling events in cardiac myocytes.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.08.004
Conversion of human cardiac progenitor cells into cardiac pacemaker-like cells.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.09.015
Shp2 in myocytes is essential for cardiovascular and neointima development.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.09.014
Intrafibrillar and perinuclear mitochondrial heterogeneity in adult cardiac myocytes.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.08.013
Homocysteine accelerates atherosclerosis by inhibiting scavenger receptor class B member1 via DNMT3b/SP1 pathway.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.11.145
The evolutionarily conserved C-terminal peptide of troponin I is an independently configured regulatory structure to function as a myofilament Ca2+-desensitizer.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.09.002
Angiotensin peptide synthesis and cyclic nucleotide modulation in sympathetic stellate ganglia
来源期刊:Journal of Molecular and Cellular CardiologyDOI:10.1016/j.yjmcc.2019.11.157
Kv4.3 expression abrogates and reverses norepinephrine-induced myocyte hypertrophy by CaMKII inhibition.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2018.11.011
Tachycardia-induced CD44/NOX4 signaling is involved in the development of atrial remodeling.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.08.006
Cytoplasmic nucleic acid-based XNAs directly enhance live cardiac cell function by a Ca2+ cycling-independent mechanism via the sarcomere.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.02.016
The lymphocyte adapter protein: A negative regulator of myocardial ischemia/reperfusion injury.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.07.003
Cardiac expression of the microsomal triglyceride transport protein protects the heart function during ischemia.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.09.003
Novel role of mitochondrial GTPases 1 in pathological cardiac hypertrophy.
来源期刊:Journal of molecular and cellular cardiologyDOI:10.1016/j.yjmcc.2019.01.025